The International Society for Evolution, Medicine, & Public Health maintains a list of over 500 people who work in areas at the interface of evolution and medicine/public health. The purpose of The Evolution and Medicine Network is to facilitate contact among those with shared interests. If, for instance, you are giving a talk in London, you can quickly find out people there who shares your interests.
If your research or teaching is focused in an area related to evolution and medicine and you would like to make it possible for others to find you please add your information to the list. It will take under a minute.
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Inexpensive generic statins, and angiotensin receptor blockers and probably other drugs (PPAR and AMPK agonists) beneficially modify the host response to inflammatory illness in many ways, including restoring endothelial barrier integrity, and thus treat or prevent ARDS/multi-organ failure in sepsis, pneumonia and influenza and GI vascular permeability in Ebola. These conditions are less frequent and less severe (lower mortality) in children than in adults (they are more tolerant of inflammatory insult), and the dividing line between the two occurs during adolescence (puberty). Why? I’ve found no studies by evolutionary biologists and very few by immunologists or clinicians that examine or explain the molecular signalling changes that distinguish pre- and post-puberty in experimental animals or humans. It seems to have been hard-wired into animals (and perhaps plants) by evolution (see Fedson DS Antiviral Res 2023: 99: 417-35). Do evolutionary biologists have ideas (e.g., life history theory) on how or why this occurs?